RXFP3 Antibody - Cat. #: CSB-PA580068

SKU: CSB-PA580068

Size :50ul

Clone Number:

Aliases:G protein coupled receptor GPCR135 antibody; G protein coupled receptor SALPR antibody; G protein-coupled receptor SALPR antibody; G-protein coupled receptor GPCR135 antibody; GPCR 135 antibody; GPCR135 antibody; Gprotein coupled receptor SALPR antibody; Relaxin 3 receptor 1 antibody; Relaxin 3 receptor1 antibody; Relaxin 3/INSL7 receptor 1 antibody; Relaxin 3/INSL7 receptor1 antibody; Relaxin family peptide receptor 3 antibody; Relaxin family peptide receptor3 antibody; Relaxin-3 receptor 1 antibody; Relaxin/insulin like family peptide receptor 3 antibody; Relaxin3 receptor 1 antibody; Relaxin3 receptor1 antibody; RL3 R1 antibody; RL3R 1 antibody; RL3R1 antibody; RL3R1_HUMAN antibody; RLN3 receptor 1 antibody; RLN3 receptor1 antibody; RLN3R1 antibody; RXFP3 antibody; RXFPR3 antibody; SALP R antibody; SALPR antibody; somatostatin and angiotensin like peptide receptor antibody; Somatostatin- and angiotensin-like peptide receptor antibody

Product Type:Polyclonal Antibody

Immunogen Species:Homo sapiens (Human)

UniProt ID:Q9NSD7

Immunogen:Synthetic peptide of Human RXFP3

Raised in:Rabbit

Species Reactivity:Human, Mouse

Tested Applications:ELISA, IHC; ELISA:1:2000-1:5000, IHC:1:25-1:100

Background:Relaxin-3 is a highly conserved neuropeptide in vertebrate species and binds to the Class A G protein-coupled receptor (GPCR) RXFP3. Relaxin-3 is involved in a wide range of behaviors and targeting of RXFP3 may be relevant for a range of neurological diseases. RXFP3, confirmed that acute and chronic activation of RXFP3 increased feeding and weight gain, and produced changes in plasma leptin and insulin.

Clonality:Polyclonal

Isotype:IgG

Purification Method:Antigen affinity purification

Conjugate:Non-conjugated

Buffer:-20°C, pH7.4 PBS, 0.05% NaN3, 40% Glycerol

Form:Liquid

Stroage:Upon receipt, store at -20°C or -80°C. Avoid repeated freeze.

Target Names:RXFP3

Research Areas:Neuroscience?Cancer;Developmental biology;Metabolism;Signal transduction