Gene Bio Systems
NDUFS6 Antibody - Cat. #: CSB-PA089578
NDUFS6 Antibody - Cat. #: CSB-PA089578
SKU:CSB-PA089578
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Size :50ul
Clone Number:
Aliases:BC059730 antibody; CI 13kA antibody; CI 13kD A antibody; CI-13kD-A antibody; CI13KDA antibody; Complex I 13kD A antibody; Complex I 13kDa subunit A antibody; Complex I mitochondrial respiratory chain 13 kD subunit antibody; Complex I-13kD-A antibody; IP13 antibody; MGC107676 antibody; NADH dehydrogenase (ubiquinone) Fe-S protein 6, 13kDa (NADH coenzyme Q reductase) antibody; NADH dehydrogenase [ubiquinone] iron sulfur protein 6, mitochondrial antibody; NADH dehydrogenase [ubiquinone] iron-sulfur protein 6, mitochondrial antibody; NADH ubiquinone oxidoreductase 13 kDa A subunit antibody; NADH-ubiquinone oxidoreductase 13 kDa-A subunit antibody; NADH:ubiquinone oxidoreductase NDUFS6 subunit antibody; NADH:ubiquinone oxidoreductase subunit S6 antibody; Ndufs6 antibody; NDUS6_HUMAN antibody
Product Type:Polyclonal Antibody
Immunogen Species:Homo sapiens (Human)
UniProt ID:O75380
Immunogen:Fusion protein of Human NDUFS6
Raised in:Rabbit
Species Reactivity:Human, Mouse, Rat
Tested Applications:ELISA, IHC; ELISA:1:2000-1:5000, IHC:1:50-1:200
Background:This gene encodes a subunit of the NADH:ubiquinone oxidoreductase (complex I), which is the first enzyme complex in the electron transport chain of mitochondria. This complex functions in the transfer of electrons from NADH to the respiratory chain. The subunit encoded by this gene is one of seven subunits in the iron-sulfur protein fraction. Mutations in this gene cause mitochondrial complex I deficiency, a disease that causes a wide variety of clinical disorders, including neonatal disease and adult-onset neurodegenerative disorders.
Clonality:Polyclonal
Isotype:IgG
Purification Method:Antigen affinity purification
Conjugate:Non-conjugated
Buffer:-20°C, pH7.4 PBS, 0.05% NaN3, 40% Glycerol
Form:Liquid
Stroage:Upon receipt, store at -20°C or -80°C. Avoid repeated freeze.
Target Names:NDUFS6
Research Areas:Cancer;Tags & Cell Markers;Metabolism;Signal transduction
